 Viral Matrix Proteins
"Viral Matrix Proteins" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Proteins associated with the inner surface of the lipid bilayer of the viral envelope. These proteins have been implicated in control of viral transcription and may possibly serve as the "glue" that binds the nucleocapsid to the appropriate membrane site during viral budding from the host cell.
| Descriptor ID |
D014763
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| MeSH Number(s) |
D12.776.964.970.880.940
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| Concept/Terms |
Viral Matrix Proteins- Viral Matrix Proteins
- Matrix Proteins, Viral
- Proteins, Viral Matrix
- Membrane Proteins, Viral
- Viral M Proteins
- M Proteins, Viral
- Proteins, Viral M
- Viral Membrane Proteins
- Proteins, Viral Membrane
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Below are MeSH descriptors whose meaning is more general than "Viral Matrix Proteins".
Below are MeSH descriptors whose meaning is more specific than "Viral Matrix Proteins".
This graph shows the total number of publications written about "Viral Matrix Proteins" by people in this website by year, and whether "Viral Matrix Proteins" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2015 | 0 | 1 | 1 | | 2017 | 1 | 0 | 1 |
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Below are the most recent publications written about "Viral Matrix Proteins" by people in Profiles.
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Kumar J, Yadav VN, Phulera S, Kamble A, Gautam AK, Panwar HS, Sahu A. Species Specificity of Vaccinia Virus Complement Control Protein for the Bovine Classical Pathway Is Governed Primarily by Direct Interaction of Its Acidic Residues with Factor I. J Virol. 2017 10 01; 91(19).
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Wen SC, Schuster JE, Gilchuk P, Boyd KL, Joyce S, Williams JV. Lung CD8+ T Cell Impairment Occurs during Human Metapneumovirus Infection despite Virus-Like Particle Induction of Functional CD8+ T Cells. J Virol. 2015 Sep; 89(17):8713-26.
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