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Srivastava, Tarak

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research overview

Tarak Srivastava, MD, Director of the Nephrology Research Laboratory at Children’s Mercy and Professor of Pediatrics, University of Missouri-Kansas City School of Medicine, supported by R01 funding from the National Institutes for Health (NIH), is making important discoveries pertaining to CKD progression, with lab research focused on podocyte cell biology, its role in the pathophysiology and potential treatment of hyperfiltration injury.His research also focuses on the role of innate immunity in idiopathic nephrotic syndrome of childhood, and in kidney development. While research on these subjects transcends into the adult world, Dr. Srivastava is one of only a handful of nephrologists focused on identifying and mitigating the risk factors for chronic kidney disease in children.

One or more keywords matched the following items that are connected to Srivastava, Tarak

Item Type	Name
Concept	Podocytes
Academic Article	Podocyte proteins in Galloway-Mowat syndrome.
Academic Article	Cell-cycle regulatory proteins in podocyte cell in idiopathic nephrotic syndrome of childhood.
Academic Article	Cell-cycle regulatory proteins in the podocyte in collapsing glomerulopathy in children.
Academic Article	LPS and PAN-induced podocyte injury in an in vitro model of minimal change disease: changes in TLR profile.
Academic Article	Fluid flow shear stress upregulates prostanoid receptor EP2 but not EP4 in murine podocytes.
Academic Article	Fluid flow shear stress over podocytes is increased in the solitary kidney.
Academic Article	Cyclooxygenase-2, prostaglandin E2, and prostanoid receptor EP2 in fluid flow shear stress-mediated injury in the solitary kidney.
Academic Article	Mechanotransduction signaling in podocytes from fluid flow shear stress.
Academic Article	Upregulated proteoglycan-related signaling pathways in fluid flow shear stress-treated podocytes.
Academic Article	Ethanol at low concentrations protects glomerular podocytes through alcohol dehydrogenase and 20-HETE.
Academic Article	Renal and Hematological Effects of CLCF-1, a B-Cell-Stimulating Cytokine of the IL-6 Family.
Academic Article	Hyperfiltration-associated biomechanical forces in glomerular injury and response: Potential role for eicosanoids.
Academic Article	Prostaglandin E(2) is crucial in the response of podocytes to fluid flow shear stress.
Academic Article	Transcription Factor ?-Catenin Plays a Key Role in Fluid Flow Shear Stress-Mediated Glomerular Injury in Solitary Kidney.

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Podocytes